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Author Topic: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.  (Read 92829 times)

mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #495 on: September 13, 2017, 18:29:22 »


https://www.ncbi.nlm.nih.gov/pubmed/27186365/ 

Spinal manipulative therapy, Graston technique® and placebo for non-specific thoracic spine pain: a randomised controlled trial.

This study indicates that there is no difference in outcome at any time point for pain or disability when comparing SMT, Graston Technique® or sham therapy for thoracic spine pain, however all groups improved with time. These results constitute the first from a fully powered randomised controlled trial comparing SMT, Graston technique® and a placebo.
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #496 on: September 17, 2017, 09:57:56 »


"Despite this hypothesis, there is currently no direct evidence that training-induced increases in muscle size contribute to training-induced increases in muscle strength."

https://www.ncbi.nlm.nih.gov/pubmed/28819744  Correlations Do Not Show Cause and Effect: Not Even for Changes in Muscle Size and Strength.
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #497 on: September 24, 2017, 22:05:13 »


ABC
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #498 on: September 25, 2017, 20:39:38 »


https://www.ncbi.nlm.nih.gov/pubmed/28850338

CONCLUSIONS: This study showed that individuals with chronic WAD and mild to moderate pain and disability, and no evidence of dysfunctional CPM, demonstrated reduced pain sensitivity, both in the cervical spine and over the tibialis anterior following an isometric, timed wall squat exercise. Cycling exercise did not increase pain sensitivity.
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #499 on: September 28, 2017, 18:43:27 »


"Bottom line is, pumping leucine into the system all the time in an attempt to drive mTOR may be a bad idea .."

https://www.news-medical.net/news/20170622/Could-the-mTOR-pathway-be-used-to-protect-against-cancer.aspx
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #500 on: October 02, 2017, 09:35:49 »


http://www.mdpi.com/2075-4663/5/4/76

Do Bodybuilders Use Evidence-Based Nutrition Strategies to Manipulate Physique?
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #501 on: October 02, 2017, 13:19:05 »

https://link.springer.com/article/10.1007/s00421-017-3725-7

Conclusions

Resistans Training leading to failure considerably increases the time needed for the recovery of neuromuscular function and metabolic and hormonal homeostasis.

Avoiding failure would allow athletes to be in a better neuromuscular condition to undertake a new training session or competition in a shorter period of time.

-------------------------------------------------------------------------------------

Jõutreening suutlikkuseni pikendab oluliselt närvisüsteemi, metaboolse- ja hormonaalse homeostaasi taastumise aega. Selle vältimine võimaldab tihedamaid treening- või võistlus-sessioone.
« Last Edit: October 02, 2017, 13:27:55 by mp »
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #502 on: October 06, 2017, 21:34:12 »


Soy isoflavones IMPROVE testosterone in diet-induced obese rodents
The rodents who were fed a high-fat high sugar diet developed obesity and signifcantly deformed seminiferous tubules compared with that in normal diet fed controls.
After receiving soy isoflavones (SIF) at doses of 0, 50, 250 and 500 mg/kg per day for four weeks, the diet-induced obese (DIO) male rats exhibited decreased body weight in a dose-dependent manner,
accompanied with signifcantly alleviated testicular damages, as well as increased testosterone levels and steroidogenic acute regulatory (StAR), cytochrome P450 11A1 (CYP11A1), cytochrome P450 17A1 (CYP17), hydroxysteroid dehydrogenase-3β (HSD3β), and hydroxysteroid dehydrogenase-17β (HSD17β) protein and mRNA levels.
"In conclusion, SIF could alleviate testicular damages, increase testosterone levels, and upregulate the expression of proteins
and genes related to testosterone synthesis in DIO male rats, which would be important for obesity male reproduction treatment," the authors write.

http://www.ijcep.com/files/ijcep0054961.pdf Effects of soy isoflavones on testosterone
synthetase in diet-induced obesity male rats
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #503 on: October 07, 2017, 19:26:36 »

RATIONALE: Severe hypokalemia can be a potentially life-threatening disorder and is associated with variable degrees of skeletal muscle weakness.
PATIENT CONCERNS: We report a case of severe hypokalemic paralysis and rhabdomyolysis in a 28-year-old bodybuilder. He was admitted to the emergency room due to progressive paralysis in both lower extremities, which had begun 12 hours earlier. He was a bodybuilder trainer and had participated in a regional competition 5 days earlier. He went on a binge, consuming large amounts of carbohydrates over 4 days, resulting in a gain of 10 kg in weight.
DIAGNOSES: He had no family history of paralysis and this was his first attack. He strongly denied drug abuse, such as anabolic steroids, thyroid and growth hormone, and diuretics. Neurological examinations revealed symmetrical flaccid paralysis in his lower extremities, but the patient was alert and his sensory system was intact. His initial serum potassium and phosphate level was 1.8 mmol/L and 1.4 mg/dL, respectively. The calculated transtubular potassium gradient (TTKG) was 2.02. His thyroid function was normal.
INTERVENTIONS AND OUTCOMES: Serum potassium levels increased to 3.8 mmol/L with intravenous infusion of about 50 mmol of potassium chloride over 20 hours.
OUTCOMES: His muscular symptoms improved progressively and he was discharged from the hospital 7 days after admission on foot. He was followed in our outpatient clinic, without recurrence.

LESSONS: Physicians should keep in mind that large intakes of food during short periods can provoke hypokalemic paralysis and rhabdomyolysis, especially in bodybuilders.

Lee TW, Bae E, Hwang K, et al. Severe hypokalemic paralysis and rhabdomyolysis occurring after binge eating in a young bodybuilder: Case report. Medicine (Baltimore) 2017;96(40):e8251.
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #504 on: October 13, 2017, 21:01:56 »


https://www.ncbi.nlm.nih.gov/pubmed/29024332

Effect of resistance training set volume on upper body muscle hypertrophy: are more sets really better than less?
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #505 on: October 14, 2017, 15:38:52 »


Brad Schoenfeld

"Slide from presentation by Dr Maarit Lehti showing a theorized hormetic response of muscle damage on muscle growth. A moderate level of damage is proposed to have a positive effect on hypertrophy but excessive damage is a negative. Take home; don't train to the point where you have difficulty walking. Stimulate, don't annihilate!"
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #506 on: October 14, 2017, 15:59:20 »


"Interesting data from a collaboration with my colleague, Alex Silva-Ribeiro. We found that one group of elite bodybuilders following a 4500 cal/day diet gained muscle without gaining any fat while another group following a 6000 cal/diet gained an extra ~1 kg of muscle but also gained an additional ~kg of fat over a 4 week period. All the extra calories came from carbohydrate; protein intake was equated between groups ~2 g/kg). The findings further support the benefit of higher caloric intakes for maximizing muscle mass - particularly in well-trained individuals - but also highlight that the gains come with the caveat of added body fat"
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #507 on: October 17, 2017, 09:43:03 »


"It has been proposed that concurrent training (doing aerobic exercise as well as strength training) produces smaller increases in muscle size compared to strength training only, because of an interference effect.
This interference effect was identified based on rodent studies, and is thought to involve AMPK phosphorylation reducing mTOR signaling. However, as this study shows, stimulating AMPK phosphorylation immediately before a strength training workout in humans does not reduce hypertrophy."
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mp

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #508 on: October 18, 2017, 11:04:35 »


"It has been proposed that concurrent training (doing aerobic exercise as well as strength training) produces smaller increases in muscle size compared to strength training only, because of an interference effect.

This important study showed that lower body aerobic exercise (either HIIT or moderate intensity steady state exercise) performed immediately before a strength training workout reduces gains in maximum lower body strength, but has no effect on gains in upper body strength.

Moreover, the reduction in lower body strength gains was similar whether performing either HIIT or steady state exercise, although there was only a reduction in hypertrophy after HIIT."
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A.L

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Re: mp siin - kui sul ei ole midagi asjalikku öelda, tee seda mujal.
« Reply #509 on: October 18, 2017, 20:07:21 »


This important study showed that lower body aerobic exercise (either HIIT or moderate intensity steady state exercise) performed immediately before a strength training workout reduces gains in maximum lower body strength, but has no effect on gains in upper body strength.

Moreover, the reduction in lower body strength gains was similar whether performing either HIIT or steady state exercise, although there was only a reduction in hypertrophy after HIIT."

Ma ju tegin esimese poole aastat 30 minutit HIIT-i rattal ja teise poole aastast väntasin 45 minutit pulsiga 120-130  ja seda ennem rasket trenni :) Ütlen ausalt, et lisandus kvaliteetset jõudu ja just sinna allapoole. Ei mitte sina, kuhu Sinu mõte kohe liikus vaid jalgadesse :) Ma räägin, teadus on kahe otsaga vorst :)

A.
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What will you do if all your problems aren't solved by the time you die?
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